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Hans Arnqvist conducts research focused on vascular metabolism, particularly in relation to diabetes and the function of insulin and IGF-I receptors. His work involves studying human endothelial cells and vascular smooth muscle cells, examining how these cells respond to insulin and IGF-I, including the presence of hybrid receptors. He has made significant observations regarding the physiological and pathophysiological importance of these receptors in relation to diabetes. Specifically, his clinical studies have revealed that circulating IGF-I levels are often 30-40% lower in type 1 diabetes patients compared to healthy controls, which is linked to glycaemic control. His research also investigates the abnormalities in the IGF-system among patients with and without endogenous insulin secretion, aiming to uncover the clinical consequences of these abnormalities. Arnqvist's current work continues to explore the interactions between insulin delivery and the IGF-system, suggesting that standard subcutaneous insulin treatment may not sufficiently regulate the IGF-system, particularly regarding its role in vascular health. His findings underscore the importance of IGF-I in vascular tissue, especially under conditions of low physiological levels.
Requirements are standardized across the Faculty of Science and Engineering (Institute of Technology) for international Master's programs.