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Nathan Woodling is a biogerontologist with a background in neuroscience and molecular genetics. He completed his PhD in 2013 at Stanford University, where he studied the way inflammatory signaling pathways in microglia contribute to the types of neurotoxicity associated with Alzheimer’s disease. Afterward, he moved to the UCL Institute of Healthy Ageing for his postdoctoral work, investigating how glial and neuronal cells modulate the aging nervous system and its signaling pathways. In 2019, he was awarded the Alzheimer’s Society Junior Fellowship, which provided him the opportunity to investigate age-related changes in glia that predispose the nervous system to the types of damage seen in Alzheimer’s disease. In 2022, Nathan relocated to Glasgow to establish his own research group, focusing on specific cell signaling pathways that modulate aging and the susceptibility to neurodegenerative diseases. The Woodling Lab aims to understand the evolutionary origins and biological underpinnings of aging, working primarily with the fruit fly Drosophila, to study the genetic factors affecting aging trajectories that are remarkably shared with humans. Current research questions include the developmental processes contributing to aging and the biological mechanisms affecting pathogenesis in age-related diseases.
University of Glasgow • Glasgow, Scotland
Leading research group focused on the biological mechanisms underlying aging and neurodegenerative diseases.