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Rebecca studied Natural Sciences at the University of Cambridge, specializing in Pharmacology, and returned to Glasgow to complete a Master of Research in Translational Medicine as part of the British Heart Foundation four-year PhD programme. During her PhD, she researched the mechanisms of ventricular arrhythmia in long QT syndrome under the supervision of Professor Rachel Myles, Professor Godfrey Smith, and Dr. Francis Burton. She currently works as a Research Associate in Professor Fuller’s lab, where her research aims to determine strategic post-translational modulation of cardiac ion channels to provide inotropic and anti-arrhythmic effects in the setting of heart failure. Rebecca is a member of the Heart Rhythm Society, the British Heart Rhythm Society, and the Physiological Society. She has been awarded the British Heart Rhythm Society Young Investigator Award. Her research interests include understanding how normal cardiac physiology is altered by disease and leveraging that knowledge to develop novel risk stratification and treatment approaches. Throughout her PhD, she developed a particular interest in the mechanisms of ventricular arrhythmia and their physiological and potential pathological implications, utilizing extensive experience with optical recordings and transmembrane voltage in Langendorff-perfused hearts and large populations of isolated cardiomyocytes to examine these mechanisms under long QT conditions. In her current role, Rebecca employs biochemical techniques and cell lines derived from human induced pluripotent stem cells to explore targeting post-translational regulation of cardiac ion channels to achieve beneficial inotropic and anti-arrhythmic effects in heart failure.
University of Glasgow • Glasgow, UK
Conduct research on cardiac ion channels and their role in heart failure.