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Richard Lewis is an Emeritus Professor of Molecular Cellular Physiology at Stanford University, where he has spent his academic career focusing on the molecular mechanisms of calcium signaling. His research primarily investigates store-operated calcium channels (SOCs) and their critical role in cellular processes. Lewis's lab has significantly advanced the understanding of how depletion of calcium from the endoplasmic reticulum triggers the opening of CRAC channels, which are essential for T cell function and immune responses. He has conducted extensive studies on the regulatory functions of STIM1 and Orai1, elucidating the interplay between the endoplasmic reticulum and plasma membrane during calcium signaling. His research contributions also include exploring the transcriptional specificity of T cells and how calcium signals influence cell fate decisions during thymocyte development. Lewis's work utilizes cutting-edge techniques such as live-cell imaging and total internal reflection fluorescence microscopy to explore these intricate signaling pathways.
Stanford University • Stanford, CA
Emeritus Professor in the Department of Molecular Cellular Physiology.
Stanford University • Stanford, CA
Responsible for overseeing graduate studies in the Department of Molecular Cellular Physiology.
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