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Samuel G. Katz graduated with a combined B.S./M.S. from Yale University in 1995 and pursued dual M.D., Ph.D. training in the Health Sciences & Technology program jointly administered by MIT and Harvard. His Ph.D. thesis, supervised by Stuart H. Orkin, focused on transcriptional regulation in hematopoiesis. Dr. Katz completed his residency in pathology and a fellowship in hematopathology at Brigham and Women’s Hospital. He conducted post-doctoral studies on apoptosis under Loren D. Walensky at the Dana-Farber Cancer Institute. Currently, he serves as an Associate Professor of Pathology at Yale University, managing a laboratory that concentrates on the intrinsic mechanisms of cell death and the ways to reprogram immune cells to mediate extrinsic programmed cell death. Dr. Katz is actively involved in patient care as a hematopathologist, studying BCL-2 proteins that govern cellular pathways and exploring therapeutic strategies to enhance apoptosis in cancer treatment. His research interests also include the determinants of immune cell survival and the optimization of immunotherapy to improve patient outcomes.
Yale University • New Haven, CT
Managing a laboratory focused on intrinsic mechanisms of cell death and reprogramming immune cells.
Administered via the Graduate School of Arts and Sciences (GSAS). GRE General is optional for PhD.