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Stephen Newland is a researcher associated with the Cambridge Immunology Network, focusing on immunology and sterile inflammation. His initial research centered on the interactions between infections and autoimmune type 1 diabetes, contributing to the hygiene hypothesis. He demonstrated that Salmonella typhimurium infection protects against the development of diabetes in NOD mice. Additionally, he played a key role in characterizing NOD ES cells and successfully generated viable, fertile mice, which facilitated the development of new tools for studying disease. Recently, he has shifted his focus to cardiovascular medicine, with interest in rare immune cells, specifically type II innate lymphoid cells, and their influence on the progression of atherosclerosis, wound healing, and myocardial infarction.
Standard postgraduate requirements for Department of Politics and International Studies (POLIS) and related humanities departments.